Figure 1738

No controlled data exist on the management of recurrent disease after transplantation. For patients with primary hyperoxaluria, measures to prevent further deposition of oxalate have proved successful in controlling recurrent renal oxalosis [9]. In diabetes mellitus, the pathophysiology of recurrent nephropathy undoubtedly reflects the same insults as those causing the initial renal failure, and good evidence exists that glycemic control can slow the development of end-organ damage. Plasma exchange and immunoadsorption are promising therapies for patients with nephrosis who have recurrent focal segmental glomerulosclerosis; however, these therapies do not provide sustained remission [6,7]. In all these cases, establishing a diagnosis of recurrent disease is critical in identifying a possible treatment modality.

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