Increased plasma BUN creatinine K etc

Bun CreatinineIncreased Bun

Figure 14 Oltguria, increase in plasmn BUN and di&itiise Ifi u'inary uffla. (Ihe patient is in ARF).

J) Decreased urinary urea, creatinine, K\ etc. (Fig. 14). Urinary urea ^ntl creatinine dtxreitse In proportion to the increase Lin plasma BLN and creatinine.

Figure 14 Oltguria, increase in plasmn BUN and di&itiise Ifi u'inary uffla. (Ihe patient is in ARF).

Figure 15 Use trw urethral cathcler only if needec and avoid unnecessarv urinary tract infections.

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Figure 15 Use trw urethral cathcler only if needec and avoid unnecessarv urinary tract infections.

PERlOn IJ

(The Kidney is in intrinsic Arutc Keual Failure (ATN)).

During this period several body systems are affected by the decrease of renal function, see an it urea se in blood concentration of toxins (BUT creatinine, middle molecules etc.). fluid overload, acidosis, clcc-trolyte imbalance and a decrease in endocrine renal funciion.

A review of the main symptoms associated with intrinsic acute renal failure follows.

During this period the patient has oliguria. However, the incidcncc of non-oliguric acute renal failure lias increased in recent years. Although. it is important to know the urine output in patients with Al M in order 10 maintain fluid balance, we have to be aware of the risk of developing urinary tract injections with the placement of urethral catheters. Therefore: we do not advise the use of urethral catheters unless it is abSOIulely necessary (Fin.

figure 16- Blood chemistry in intrinsic acute renal Is ¡lure.

Stood shown (Fig. 16)

Increase in bUN and creatinine increase In uric acid

Decrease in pH (acidosis)

Hyponatremia

Hyperkalemia

Hypocalcemia

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